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Hypertension may occur in hospitalized
SARS-CoV-2 patients, and could become a sequela of COVID-19 infection.
According to a recent study in PloS One, hypertension or
high blood pressure (BP), occasionally accompanied by raised cardiac troponin I (cTnI)
may arise in COVID-19 infected individuals. Also,
augmenting Ang II signaling might play an pivotal role in development of
hypertension in COVID-19.
Cardiovascular impairment has been frequently observed
in COVID-19 patients. Ganxiao Chen and colleagues examined the consequence of
COVID-19 infection on cardiac injury and high BP.
The clinical data of total of 366 critically-ill
COVID-19 patients were analyzed and reviewed. Out of 268 patients, 30 patients (11.19%) patients
reported cardiac impairment. Out of these, 93.33% (28/30) of cases were of
severe intensity. The neutrophils, white blood
cells
(WBCs), procalcitonin,
C-reactive protein (CRP levels), lactate, and lactic dehydrogenase were
certainly linked with cardiac injury marker as per the lab
findings.
One hundred ninety patients without history
of high BP had higher angiotensin-II (AngⅡ). Out of these
patients, 16 (8.42%) patients had a rise in BP in accordance with the
diagnostic standards of hypertension during hospital stay, with a considerably
augmented level of the cTnI, procalcitonin, AngⅡ compared to those with normal
BP.
The clinical
disease severity of critically-ill COVID-19 patients can be recognized via a
predictive model, founded on the 4 predictors (older age, higher cTnI; history of high
BP, and diabetes) along with gender, the study investigators declared.
PloS One
Hypertension as a sequela in patients of SARS-CoV-2 infection
Ganxiao Chen et al.
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